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THESIS TOPICS OF MDS ORTHODONTICS IN BPKIHS DONE BY MDS POST GRADUATE RESIDENTS

 This is a compilation of all the thesis topics done at B.P. Koirala Institute of Health Sciences, Dharan, Nepal, by postgraduate residents in the Department of Orthodontics and Dentofacial Orthopedics.

1. ORIENTATION AND POSITION OF HYOID BONE IN ORTHODONTIC PATIENTS WITH DIFFERENT DENTOFACIAL GROWTH PATTERNS REPORTING TO CODS, BPKIHS: Dr. Bhushan Bhattarai, July 2014

2. THE SHAPE, SIZE, AND BRIDGING OF THE SELLA TURCICA IN SKELETAL CLASS I, II & III ADULT ORTHODONTIC PATIENTS, REPORTING TO CODS, BPKIHS: Dr. Gunjan Kumar Shrestha, July 2014

3. PHARYNGEAL WIDTHS COMPARISON IN DIFFERENT SKELETAL MALOCCLUSIONS AND GROWTH PATTERNS OF PATIENTS VISITING DEPARTMENT OF ORTHODONTICS, BPKIHS: Dr. Nabin Kumar Chaudhary, December 2020

4. CORRELATION BETWEEN DENTAL ARCH WIDTH AND VERTICAL FACIAL MORPHOLOGY IN PATIENTS SEEKING ORTHODONTIC TREATMENT IN BPKIHS, DHARAN, NEPAL: Dr. Subash Shrestha, May 2023

5. NEPALESE TRANSLATION AND VALIDATION OF THE ORAL AESTHETIC SUBJECTIVE IMPACT SCORE (OASIS) QUESTIONNAIRE: Dr. Sushant Pandey, June 2024

6. CORRELATION OF CRANIOFACIAL MEASUREMENTS ON A STANDARDIZED LATERAL PHOTOGRAPH WITH ANALOGOUS MEASUREMENTS ON A LATERAL CEPHALOGRAM: Dr. Raman Dhungel, August 2025

More topics will be updated soon.

MDS Orthodontics - Thesis Topics

 These are some very useful thesis topics that are practically useful and doable by postgraduate students.

## 1. Cephalometric and Radiographic Studies cephalometric analysis in orthodontic treatment planning

These studies utilize 2D radiographs (Lateral Cephalograms and OPGs) which are widely available.

  1. Cephalometric Norms for a Specific Nepalese Ethnic Group: Establish Steiner's or Tweed's cephalometric norms for a specific population (e.g., Newar, Rai, Gurung) to create a local database.

    • Practical Application: Provides more accurate diagnostic standards for local populations instead of relying solely on Caucasian norms.

  2. Soft Tissue Profile Changes after First Premolar Extractions: A retrospective study evaluating changes in the lips and chin position in Class I bimaxillary protrusion cases.

    • Practical Application: Helps in predicting esthetic outcomes and managing patient expectations regarding facial changes.

Navigating Advanced Dental Education: Fellowships, Clerkships, and Specializing in Cleft Lip and Palate Orthodontics

I. Introduction to Advanced Dental Training
This report provides a comprehensive overview of advanced educational pathways within the medical and dental fields, specifically distinguishing between fellowships and clerkships. The primary aim is to offer detailed guidance for orthodontists holding a Master of Dental Surgery (MDS) degree who seek to pursue further specialization in cleft lip and palate orthodontics. The information presented herein is designed to equip dental professionals with the knowledge necessary to make informed decisions regarding their advanced training and career trajectory.

Your specific interest in cleft lip and palate orthodontics underscores a desire for highly specialized, multidisciplinary training that extends beyond a general orthodontic residency. This report will address the unique requirements, opportunities, and challenges inherent in this niche field, providing a structured pathway for aspiring specialists.

II. Understanding Fellowships in Medical and Dental Fields
A. What is a Fellowship?
A fellowship represents a period of highly specialized medical or dental training undertaken after the successful completion of a core specialty training program, such as a residency. In the United States and Canada, during this advanced training period, the individual is formally known as a "fellow" or "fellow physician". For dentists, a fellowship typically constitutes a post-residency experience with a concentrated focus on a very specific area of practice, such as oral and maxillofacial surgery or, pertinently for this report, craniofacial orthodontics.

It is important to distinguish this clinical training fellowship from honorary fellowships, such as a "Fellowship in the Academy of Dentistry International".While the latter is a distinctive honor bestowed upon individuals for outstanding accomplishments and contributions to the dental profession (e.g., in clinical practice, research, education, or public service), it does not represent a period of structured clinical or research training in a subspecialty. The focus of this report is exclusively on the clinical and research-oriented training fellowships that lead to advanced specialization.

B. Purpose and Benefits of Fellowships
The fundamental purpose of a medical or dental fellowship is to cultivate expert practitioners within highly specialized subspecialties.2 Fellowships serve as a crucial transitional phase, enabling a resident to evolve into a fully independent specialist.2 This period is invaluable for forming professional connections and accessing advanced job opportunities within the chosen subfield.

Fellowship programs are characterized by their provision of significant, practical work experience, often granting fellows a substantial degree of responsibility early in their training. During this time, fellows collaborate closely with seasoned specialists, allowing them to profoundly deepen their clinical experience and knowledge within their specific subspecialty. Upon the successful completion of a fellowship program, the physician or dentist earns the esteemed title of "fellowship-trained." This designation signifies the highest level of dedication to their chosen field and qualifies them to practice medicine or dentistry independently within their subspecialty without direct supervision. Beyond direct patient care, achieving fellowship-trained status can enhance patient trust and open avenues for future leadership roles, including the opportunity to train subsequent generations of fellows.

C. Typical Duration and Structure of Fellowships
The typical duration of a medical or dental fellowship varies, generally ranging from one to three years, depending on the specific subspecialization. For instance, many craniofacial orthodontics fellowships are structured as 12-month programs. These programs are designed to provide advanced training within a highly focused subspecialty, with the explicit aim of refining and deepening a practitioner's expertise and skills beyond the scope of their initial residency training. The structure of fellowships consistently integrates several key components: intensive clinical training, comprehensive didactic instruction, and dedicated research activities. Fellows engage in hands-on patient care, participate in specialized conferences and seminars, and often undertake a significant research project to contribute to the scientific body of knowledge in their subspecialty.

III. Understanding Clerkships in Medical and Dental Fields

MCQs in Orthodontics - Orthodontic Growth and Development Assessment


# Which of the following terms describes an increase in the number of cells?
A. Hypertrophy
B. Maturation
C. Accretion
D. Hyperplasia

# The concept that growth occurs along an axis from head to tail is known as which growth pattern?
A. Anteroposterior
B. Proximodistal
C. Somatic
D. Cephalocaudal

# According to Scammons' curves, which tissue system exhibits the most rapid growth during the first few years of life, followed by a plateau?
A. Genital system
B. General body growth
C. Lymphoid system
D. Neural system

# Which theory of craniofacial growth posits that growth occurs primarily due to adaptive responses of the skeletal tissues to functional demands of surrounding soft tissues and spaces?
A. Sutural theory
B. Servosystem theory
C. Cartilage theory
D. Functional Matrix Theory

# The primary mechanism for the increase in size of the cranial vault after birth is:
A. Surface remodeling on the external surface of the vault
B. Endochondral ossification at the cranial base
C. Appositional growth at the condylar cartilage
D. Intramembranous bone formation at the sutures

# Which cranial base synchondrosis typically fuses last, continuing to contribute to anteroposterior cranial base growth into late adolescence?
A. Pterygomaxillary synchondrosis
B. Spheno-ethmoidal synchondrosis
C. Intersphenoid synchondrosis
D. Spheno-occipital synchondrosis

# Forward and downward growth of the maxilla primarily occurs through:
A. Appositional growth at the alveolar processes
B. Endochondral ossification within the maxilla itself
C. Direct bone formation at the midpalatal suture
D. Bone deposition at the maxillary sutures and surface remodeling

# Which of the following is the primary mechanism for the growth of the mandible during childhood?
A. Intramembranous ossification of the corpus
B. Endochondral ossification at the condylar cartilage
C. Growth at the symphysis
D. Sutural growth at the posterior border of the ramus

# According to Lavergne and Gasson's classification, which type of mandibular rotation refers to the rotation of the basal bone relative to the cranial base?
A. Intramatrix rotation
B. Matrix rotation
C. Total rotation
D. Alveolar rotation

# A patient with a high Frankfort-Mandibular Plane Angle (FMA) and a tendency towards an anterior open bite often exhibits which of Tweed's growth patterns?
A. Type A (Average/Mesofacial)
B. Type B (Horizontal/Brachyfacial)
C. Type C (Vertical/Dolichofacial)
D. Type D (Unclassified)

# Which pharyngeal arch gives rise to the muscles of mastication and the maxilla/mandible?
A. First pharyngeal arch
B. Second pharyngeal arch
C. Third pharyngeal arch
D. Fourth pharyngeal arch

# The primary palate develops from the fusion of which embryonic structures?
A. Two lateral palatine processes
B. Nasal septum and palatal shelves
C. Frontonasal prominence and mandibular prominences
D. Two medial nasal prominences and two maxillary prominences

# At what approximate gestational age does the fusion of the secondary palate typically begin?
A. Week 4
B. Week 6
C. Week 7-8
D. Week 10

# The primary driving force for the elevation of the palatal shelves during secondary palate formation is widely attributed to:
A. Rapid bone growth within the shelves
B. Tongue growth pushing the shelves upward
C. Intrinsic mesenchymal turgor pressure due to hyaluronic acid accumulation
D. Muscle contractions within the shelves

# Programmed cell death (apoptosis) plays a crucial role in the fusion of the palatal shelves by eliminating which specific tissue?
A. Nasal septal cartilage
B. Medial edge epithelium (MEE)
C. Mesenchymal cells of the shelves
D. Oral epithelial cells on the superior surface

# Which of the following maternal conditions during pregnancy is a known environmental factor that increases the risk of cleft palate?
A. Maternal anemia
B. Use of certain anticonvulsant medications (e.g., phenytoin)
C. Maternal hypothyroidism
D. Excessive intake of Vitamin C

# The neonatal line in enamel and dentin is formed as a result of:
A. Physiological stress associated with birth
B. Genetic predisposition for enamel hypoplasia
C. Trauma during eruption of primary teeth
D. Pre-natal systemic illness

# Which of the following conditions would typically lead to a thinner or less distinct neonatal line?
A. Diabetic mother
B. Low birth weight
C. Elective Caesarean section delivery
D. Asphyxia in the newborn

MCQs on Cephalometric Parameters and Malocclusion


# Which of the following cephalometric parameters suggest skeletal class III malocclusion? 1. ANB is less than 0 degree 2. SNA= 84 degrees 3. SNB=82 degrees 4. Wit’s appraisal shows point A before point B 5. FMA is 40 degrees
A. 2, 4 and 5
B. 1, 4 and 5
C. 3, 4 and 5
D. 1 and 3

# A patient presents with anterior crowding as a result of large size of teeth in comparison to the base of mandible. This will be classified as:
A. Tertiary crowding
B. Secondary crowding
C. Third degree crowding
D. Primary crowding

# Which of the following cephalometric angles is primarily used to assess the anteroposterior position of the maxilla relative to the cranial base?
A. FMA
B. SNB
C. ANB
D. SNA

# A patient with a skeletal Class II malocclusion typically exhibits which of the following characteristics?
A. Prognathic mandible
B. ANB angle greater than 4°
C. Retrognathic maxilla
D. Point B anterior to Point A on Wit's appraisal

# Which cephalometric parameter assesses the vertical relationship between the Frankfort Horizontal plane and the mandibular plane?
A. FMA
B. Y axis
C. Facial angle
D. SN-GoGn

# What is the normal average value for the SNA angle in a Caucasian population?
A. 86°
B. 78°
C. 82°
D. 90°

# A high FMA angle (e.g., 30° or more) is often associated with which facial growth pattern?
A. Anteroposterior growth pattern
B. Horizontal growth pattern
C. Vertical growth pattern
D. Neutral growth pattern

# Which of the following describes a normal skeletal Class I relationship according to cephalometric analysis?
A. Wit's appraisal with point B anterior to point A
B. ANB angle between 0° and 4°
C. ANB angle of 5°
D. SNA=80° and SNB=76°

Which of the following cephalometric parameters suggest skeletal class III malocclusion?

 # Which of the following cephalometric parameters suggest skeletal class III malocclusion?
1. ANB is less than 0 degree
2. SNA= 84 degrees
3. SNB=82 degrees
4. Wit’s appraisal shows point A before point B
5. FMA is 40 degrees (INICET 2025)
A. 1, 4 and 5
B. 1 and 3
C. 2, 4 and 5
D. 3, 4 and 5



The correct answer is B. 1 and 3. 

Explanation:

  • 1. ANB is less than  A negative ANB angle indicates that point B (mandible) is anterior to point A (maxilla), which is a classic sign of skeletal Class III malocclusion.

  • 3. SNB=: While the average SNB is around 80 degrees, an SNB of 82 degrees indicates a more protrusive mandible relative to the cranial base, which is consistent with a Class III tendency (especially if SNA is normal or reduced). In contrast, SNA=84 degrees (option 2) suggests a protrusive maxilla, which is typically seen in Class II, not Class III.

  • 4. Wit’s appraisal shows point A before point B: This indicates a Class II skeletal relationship, where the maxilla is anterior to the mandible. For Class III, point B would be anterior to point A.

  • 5. FMA is : FMA (Frankfort Mandibular Plane Angle) indicates the vertical growth pattern. A high FMA (40 degrees is significantly high) suggests a vertical growth pattern or an open bite tendency, but it does not directly indicate a sagittal Class III relationship.

A patient presents with anterior crowding as a result of large size of teeth in comparison to the base of mandible. This will be classified as:

 # A patient presents with anterior crowding as a result of large size of teeth in comparison to the base of mandible. This will be classified as: (AIIMS PG 2020)
A. Primary Crowding
B. Secondary Crowding
C. Tertiary crowding
D. Third degree crowding




The correct answer is A. Primary Crowding.

Explanation:

Primary Crowding refers to crowding that arises due to a disproportion between the size of the teeth and the size of the jawbones. In this case, the patient has large teeth relative to a smaller mandible, leading to anterior crowding. This is often genetic in origin and present even before all permanent teeth have erupted.

Secondary Crowding typically develops later, often due to factors like premature loss of primary teeth, leading to mesial migration of posterior teeth and a reduction in arch length for the erupting permanent teeth.

Tertiary Crowding (also known as late adult crowding) usually occurs in late adolescence or early adulthood, often involving the lower anterior teeth, and its exact etiology is multifactorial but can involve late mandibular growth, mesial drift, and eruption of third molars.

Third-degree crowding is not a standard classification of crowding etiology; rather, crowding is often quantified as mild, moderate, or severe, sometimes with degrees (e.g., in millimeters of discrepancy), but not as "first, second, or third degree" in terms of cause. 

Anterior inclined plane (Catlan’s appliance) cannot be used in the following cases EXCEPT:

 # Anterior inclined plane (Catlan’s appliance) cannot be used in the following cases EXCEPT:
A. Posterior open bite
B. Class I with anterior flaring
C. Severe class III with reverse overjet
D. Anterior open bite

The correct answer is A. Posterior open bite.

The Catlan's appliance's mechanism of action involves disoccluding the posterior teeth, which is functionally similar to a posterior open bite. Therefore, an existing posterior open bite does not inherently prevent the appliance from being used to correct a co-existing anterior crossbite.

The most common contraindications for Catlan's appliance are:

  1. Existing anterior open bite

  2. Periodontally compromised mandibular anterior teeth

  3. Cases where further proclination of maxillary anterior teeth is undesirable (e.g., severe anterior flaring if the goal is to reduce it).

  4. Severe skeletal discrepancies.

Comparing the options:

  • A. Posterior open bite: Not a direct contraindication for using Catlan's, but Catlan's doesn't treat it and could lead to anterior open bite.

  • B. Class I with anterior flaring: Catlan's causes flaring, so contraindication if flaring is the problem.

  • C. Severe class III with reverse overjet: Catlan's is not for severe skeletal problems.

  • D. Anterior open bite: Strong contraindication.

The question is effectively asking for the case where it can be used. If the question implies that the appliance is generally contraindicated in all but one of the listed situations.

Given that Catlan's appliance creates an anterior bite plane, which effectively creates a temporary posterior open bite to allow the anterior crossbite correction, a pre-existing posterior open bite is not an absolute contraindication in the same way an anterior open bite is. It simply means the appliance isn't addressing the posterior open bite.

Therefore, the most logical answer as the "EXCEPT" case (where it can be used, or is not contraindicated) is A.

Salary Analysis of Bachelor of Dental Surgery (BDS) Graduates and General Dentists Worldwide

 

Salary Analysis of Bachelor of Dental Surgery (BDS) Graduates and General Dentists Worldwide

The Bachelor of Dental Surgery (BDS) degree is a professional undergraduate program that prepares graduates to become licensed dentists, focusing on oral health, diagnosis, and treatment of dental conditions. General dentists, typically BDS graduates or those with equivalent qualifications like Doctor of Dental Surgery (DDS) or Doctor of Dental Medicine (DMD), form the backbone of dental care worldwide. This article provides a comprehensive overview of the salaries of BDS graduates and general dentists across various countries, highlighting average, top, and minimum salaries, and the factors influencing these earnings.



Overview of BDS Graduates and General Dentists

BDS graduates are trained to perform a wide range of dental procedures, including cleanings, fillings, extractions, and patient education on oral health. After completing their degree, they may work as general dentists in private practices, public hospitals, or academic institutions, or pursue further specialization through programs like Master of Dental Surgery (MDS). Salaries for BDS graduates and general dentists vary significantly based on factors such as geographic location, experience, work setting (public vs. private), and additional qualifications.

The global demand for dental professionals is rising due to increased awareness of oral health, an aging population, and advancements in dental technology. However, salary disparities exist across countries due to differences in economic conditions, healthcare systems, and cost of living. Below, we explore the salary landscape for BDS graduates and general dentists worldwide, with specific data for key countries where available.

Periodontology MCQs - Flap and Mucogingival Surgery


# Granulation tissue is replaced by connective tissue in:
A. 7 days
B. 14 days
C. 21 days
D. 1 month

# In guided tissue regeneration technique for root coverage, the titanium reinforced membrane was used to create space beneath the membrane by:
A. Tinti and Vincenzi
B. Pini Prato and Tonetti
C. Ramjford and Nissle
D. Widman and Cohen

Primary immunoglobulin secreted or activated after vaccination:


# Primary immunoglobulin secreted or activated after vaccination:
A. IgM
B. IgA
C. IgG
D. IgE


The primary immunoglobulin secreted or activated after vaccination is:

C. IgG

Explanation:
IgG is the most abundant antibody in blood and extracellular fluid, making up ~75% of serum immunoglobulins. It is the key effector of the secondary immune response (activated after initial exposure or vaccination).
IgM is produced first during the primary immune response but is short-lived. Vaccination typically aims to induce long-term immunity via IgG.
IgA is important for mucosal immunity (e.g., respiratory/gut lining) but is not the dominant systemic response to vaccines.
IgE is associated with allergies/parasitic infections and plays no significant role in vaccine-induced immunity.

According to Clark’s rule for localization of object, if tube is shifted mesially to original angulation:

# According to Clark’s rule for localization of object, if tube is shifted mesially to original angulation:
A. Buccal objects move mesially
B. Lingual objects move mesially
C. Both buccal and lingual objects move distally
D. Both buccal and lingual objects move mesially


Correct answer: B. Lingual objects move mesially

According to Clark’s rule (also known as the SLOB rule—Same Lingual, Opposite Buccal) for object localization in dental radiography, when the X-ray tube is shifted mesially while maintaining the original angulation:

Buccal objects (those closer to the cheek) move in the opposite direction of the tube shift, so they move distally.
Lingual objects (those closer to the tongue) move in the same direction as the tube shift, so they move mesially.

Given the options:
A. Buccal objects move mesially: Incorrect, as buccal objects move distally.
B. Lingual objects move mesially: Correct, as lingual objects follow the tube shift direction.
C. Both buccal and lingual objects move distally: Incorrect, as lingual objects move mesially.
D. Both buccal and lingual objects move mesially: Incorrect, as buccal objects move distally.

All of the following are the features of dysplasia EXCEPT:

# All of the following are the features of dysplasia EXCEPT: 
A. Enlarged nuclei and cells
B. Increased nuclear to cytoplasmic ratio
C. Hypochromatic nuclei
D. Pleomorphic nuclei and cells


The correct answer is C. Hypochromatic nuclei.

Explanation: Dysplasia is characterized by abnormal cellular changes, including enlarged nuclei and cells, increased nuclear to cytoplasmic ratio, and pleomorphic nuclei and cells (variation in size and shape). These are all hallmark features of dysplasia. However, hypochromatic nuclei (nuclei with reduced staining, appearing pale) are not typically associated with dysplasia. Instead, dysplastic cells often have hyperchromatic nuclei (darkly staining due to increased DNA content), which is a key feature of the condition.

Renin secretion is stimulated by all EXCEPT:

# Renin secretion is stimulated by all EXCEPT:
A. Cardiac failure
B. Low Na+ in proximal tubule
C. Sympathetic stimulation
D. High Na+ in proximal tubule


The correct answer is D. High Na+ in proximal tubule.

Explanation: Renin secretion, primarily by the juxtaglomerular cells in the kidneys, is stimulated by factors that signal a need to increase blood pressure or sodium retention. These include:

A. Cardiac failure: Reduced cardiac output lowers renal perfusion, stimulating renin release to activate the renin-angiotensin-aldosterone system (RAAS) to restore blood pressure.
B. Low Na+ in proximal tubule: Detected by the macula densa, low sodium levels signal reduced filtrate delivery, triggering renin secretion to promote sodium reabsorption and increase blood volume.
C. Sympathetic stimulation: Activation of the sympathetic nervous system, via beta-adrenergic receptors, directly stimulates renin release to address stress or low blood pressure.
D. High Na+ in proximal tubule, however, does not stimulate renin secretion. High sodium levels in the proximal tubule (or at the macula densa in the distal tubule) typically indicate adequate or excessive sodium delivery, suppressing renin release as there is no need to activate RAAS.

As no dental biomaterial is absolutely free from the potential risk of adverse reactions, the testing of biocompatibility is related to:

 # As no dental biomaterial is absolutely free from the potential risk of adverse reactions, the testing of biocompatibility is related to:
A. Risk factors
B. Risk assessment
C. Risk markers
D. Risk predictors




The correct answer is B. Risk assessment.

Explanation: Biocompatibility testing for dental biomaterials focuses on evaluating the potential for adverse biological reactions, such as toxicity or irritation, when the material interacts with the body. This process is a key component of risk assessment, which involves systematically identifying, analyzing, and evaluating risks to ensure the material is safe for use. By assessing these risks, manufacturers and clinicians can minimize potential harm to patients.

Traumatic injury of a nerve causing paresthesia is:

 # Traumatic injury of a nerve causing paresthesia is:
A. Neuropraxia
B. Neurotmesis
C. Axonotmesis
D. Toxolysis


The correct answer is A. Neuropraxia.

Explanation:

  • Neuropraxia is the mildest form of traumatic nerve injury, involving a temporary conduction block due to compression or mild trauma. It often causes paresthesia (tingling or numbness) without significant structural damage to the nerve. Recovery is usually complete within days to weeks.
  • Neurotmesis is the most severe nerve injury, involving complete nerve transection with disruption of the nerve and its sheath, leading to permanent loss of function unless surgically repaired. Paresthesia may occur, but it’s not the primary feature.
  • Axonotmesis involves damage to the axons but preservation of the nerve’s connective tissue. It causes more severe symptoms than neuropraxia, with longer recovery times (weeks to months), and paresthesia may be present but is less characteristic.
  • Toxolysis is not a standard term in nerve injury classification and is incorrect in this context.

Since the question specifies a traumatic nerve injury causing paresthesia (a sensory symptom like tingling), neuropraxia is the most fitting answer due to its association with mild, reversible sensory disturbances.


A tumor characterized by rapid rate of growth which almost doubles its size by next day:

 # A tumor characterized by rapid rate of growth  which almost doubles its size by next day:
A. Hodgkin’s Lymphoma
B. Malignant melanoma
C. African Burkitt’s jaw lymphoma
D. Squamous cell carcinoma


The correct answer is C. African Burkitt’s jaw lymphoma.

African Burkitt’s lymphoma, particularly the endemic form, is characterized by an extremely rapid growth rate, with tumors often doubling in size within 24 hours. This aggressive B-cell non-Hodgkin lymphoma is commonly seen in children in equatorial Africa and is strongly associated with Epstein-Barr virus (EBV). It frequently presents as a rapidly enlarging jaw or facial mass. While Hodgkin’s lymphoma, malignant melanoma, and squamous cell carcinoma can be aggressive, they do not typically exhibit such an exceptionally rapid doubling time as seen in Burkitt’s lymphoma.

Popsicle panniculitis can occur:

 # Popsicle panniculitis can occur:
A. Following prolonged exposure to analgesic powder
B. Following prolonged exposure to hot beverages
C. Following prolonged exposure to frozen food
D. Following prolonged exposure to a sharp tooth


The correct answer is C. Following prolonged exposure to frozen food.

Popsicle panniculitis is a form of cold-induced panniculitis, an inflammation of the subcutaneous fat, typically seen in infants or young children. It occurs due to prolonged exposure to cold objects, such as frozen food (e.g., popsicles), which can cause localized fat necrosis and inflammation. The condition is often seen on the cheeks or chin after contact with cold items. The other options—analgesic powder, hot beverages, or a sharp tooth—are not associated with this condition. 

The most common pathogens responsible for nosocomial pneumonias in the ICU are:

 # The most common pathogens responsible for nosocomial pneumonias in the ICU are:
A. Gram positive organisms
B. Gram negative organisms
C. Mycoplasma
D. Virus infections


The correct answer is B. Gram negative organisms.

Gram-negative bacteria, such as Pseudomonas aeruginosa, Klebsiella pneumoniae, and Acinetobacter species, are the most common pathogens responsible for nosocomial (hospital-acquired) pneumonias in the ICU. These organisms are often associated with ventilator-associated pneumonia (VAP) and thrive in the ICU environment due to factors like prolonged mechanical ventilation, invasive procedures, and antibiotic resistance. Gram-positive organisms (e.g., Staphylococcus aureus) are less common but still significant, while Mycoplasma and viral infections are rare causes of ICU-acquired pneumonia.


Chicken fat clot is:

# Chicken fat clot is:
A. Postmortem clot
B. Thrombus
C. Infarct
D. None of the above


The correct answer is A. Postmortem clot.

A chicken fat clot is a type of postmortem clot, typically seen in autopsies. It forms after death due to the settling of red blood cells and plasma, creating a layered appearance with a yellowish "chicken fat" layer of plasma and fibrin on top and a darker red blood cell layer below. Unlike a thrombus, which forms in living tissue and can obstruct blood flow, a chicken fat clot occurs post-mortem and is not associated with disease processes like infarction.

Therapy of choice for pockets with an edematous wall is:

 # Therapy of choice for pockets with an edematous wall is:
A. Scaling and root planing
B. Gingivectomy
C. Apically displaced flap
D. Root resection


The correct answer is A. Scaling and root planing.

This non-surgical procedure is typically the first line of treatment for periodontal pockets with edema, as it addresses the underlying inflammation and infection by removing plaque, tartar, and bacterial toxins from the tooth surfaces and root, promoting healing and reduction of pocket depth.

Paederus Dermatitis: What You Need to Know About This Painful Insect Reaction - Acid Fly - Nairobi Fly Dermatitis

 

Paederus Dermatitis: What You Need to Know About This Painful Insect Reaction

Introduction

Paederus dermatitis, also known as "rove beetle dermatitis" or "Nairobi fly dermatitis," is a painful and often alarming skin condition caused by contact with certain species of rove beetles, particularly those from the Paederus genus. These small insects don’t bite or sting, but their body fluids contain a potent toxin called pederin, which can cause severe skin irritation. I recently experienced this myself, resulting in a large, scary wound that prompted me to raise awareness about this little-known but dreadful insect. This article will explain what Paederus dermatitis is, its symptoms, causes, treatment, and prevention strategies to help you avoid the same discomfort.

Day 3 Crusting and healing of wound

Day 2 After contact with the Acid Fly


What is Paederus Dermatitis?

Paederus dermatitis is a type of irritant contact dermatitis caused by crushing a Paederus beetle against the skin. These beetles, often mistaken for ants or small flies, are typically 7–10 mm long, with a distinctive appearance featuring black and orange or red markings. They are commonly found in tropical and subtropical regions, including parts of Africa, Asia, South America, and Australia, often in warm, humid environments near agricultural fields or rivers.

Unlike bites or stings, the reaction occurs when the beetle’s hemolymph (body fluid), which contains pederin, comes into contact with the skin. This toxin is highly irritating and can cause painful, burning lesions that may last for days or weeks if not properly managed.

Symptoms of Paederus Dermatitis

The symptoms of Paederus dermatitis typically appear 12–48 hours after contact with the beetle and can vary in severity. Common symptoms include:

  • Redness and Burning Sensation: The affected area becomes red and feels intensely warm or burning.
  • Blisters and Lesions: Small blisters or pustules form, often progressing to larger, open sores or ulcers.
  • Itching and Pain: The lesions are often itchy and painful, making it difficult to ignore.
  • Linear Marks: The lesions may appear in streaks or linear patterns, as the beetle is often unknowingly brushed across the skin.
  • Secondary Infections: Scratching the affected area can introduce bacteria, leading to infections that worsen the condition.

In my case, I developed a large, red, and painful wound that looked alarming and took weeks to heal fully. The delayed onset of symptoms can make it hard to connect the reaction to the beetle, as many people don’t recall encountering the insect.



Causes and Risk Factors

Paederus beetles are attracted to artificial lights, which often brings them into homes or outdoor areas at night. Crushing the beetle, either intentionally or accidentally (e.g., swatting it or brushing it off the skin), releases pederin, triggering the dermatitis. Key risk factors include:

  • Geographic Location: Living in or visiting tropical or subtropical regions where these beetles are prevalent.
  • Seasonal Factors: Outbreaks are more common during rainy seasons when beetle populations increase.
  • Nighttime Exposure: The beetles are nocturnal and drawn to lights, increasing the risk of contact in the evening.
  • Unawareness: Many people, including myself, are unaware of the beetle’s danger and may crush it, worsening the exposure.

Treatment and Management

If you suspect Paederus dermatitis, prompt action can reduce the severity of symptoms. Here’s what to do:

  1. Wash the Area Immediately: If you believe you’ve come into contact with a beetle, wash the affected area with soap and water to remove any residual pederin. This can help minimize the reaction if done quickly.
  2. Avoid Scratching: Scratching can worsen the lesions and lead to secondary infections.
  3. Apply Cold Compresses: A cold compress can reduce burning and inflammation.
  4. Use Topical Treatments: Over-the-counter hydrocortisone cream or antihistamines can help with itching and inflammation. For severe cases, consult a doctor for stronger topical or oral steroids.
  5. Monitor for Infection: If the wound shows signs of infection (e.g., increased redness, swelling, or pus), seek medical attention. Antibiotics may be needed.
  6. Keep the Area Clean and Dry: Proper hygiene can prevent complications and promote healing.

In my experience, washing the area and using a mild corticosteroid cream helped, but the wound still took time to heal. Consulting a healthcare professional early can make a significant difference.

Prevention Tips

Preventing Paederus dermatitis requires awareness and caution, especially in areas where these beetles are common. Here are some practical tips:

  • Avoid Crushing Beetles: If you see a small black-and-orange insect, gently brush it off or blow it away rather than crushing it.
  • Use Insect Screens: Install fine mesh screens on windows and doors to keep beetles out, especially at night.
  • Limit Light Exposure: Turn off unnecessary outdoor lights or use yellow bulbs, which are less attractive to beetles.
  • Wear Protective Clothing: Long sleeves and pants can reduce skin exposure when outdoors in beetle-prone areas.
  • Check Bedding and Clothing: Shake out clothes, towels, or bedding before use, as beetles may hide in fabrics.
  • Stay Informed: Learn to recognize Paederus beetles and their habitats, especially if you live in or travel to affected regions.

Conclusion

Paederus dermatitis is a painful and distressing condition that can catch anyone off guard, as it did me when I developed a large, alarming wound after unknowingly crushing a rove beetle. By understanding the causes, symptoms, and prevention strategies, you can protect yourself and your loved ones from this dreadful insect. If you suspect contact with a Paederus beetle, act quickly to minimize the reaction and seek medical advice if symptoms worsen. Awareness is key—stay vigilant, especially in warm, humid environments, and share this knowledge to help others avoid the same ordeal.

Temporary Anchorage Devices in Orthodontics: A Patient’s Guide

 

Temporary Anchorage Devices in Orthodontics: A Patient’s Guide

If you’re undergoing orthodontic treatment, you may have heard your orthodontist mention Temporary Anchorage Devices (TADs). These small, innovative tools have revolutionized modern orthodontics, offering precise and efficient solutions for complex tooth movements. This article explains what TADs are, how they work, their benefits, and what you can expect if they’re part of your treatment plan.

What Are Temporary Anchorage Devices (TADs)?

Temporary Anchorage Devices, or TADs, are small, screw-like devices made of biocompatible materials, such as titanium, that are temporarily placed in the jawbone to assist with orthodontic treatment. Think of them as stable anchors that provide a fixed point for moving teeth in ways that traditional braces or aligners alone might not achieve.

TADs are typically 6–12 mm long and about 1–2 mm in diameter, similar in size to a small earring post. They’re placed in specific areas of the mouth by an orthodontist or oral surgeon and removed once they’ve served their purpose.



How Do TADs Work?

Orthodontic treatment often involves applying controlled forces to move teeth into their desired positions. TADs act as anchors to support these forces, ensuring that only the targeted teeth move while others stay in place. Here’s a simple breakdown of how they work:

  1. Placement: The orthodontist numbs the area with local anesthesia and gently inserts the TAD into the bone through the gum tissue. This is a quick, minimally invasive procedure, often taking just a few minutes.
  2. Anchoring: Once in place, the TAD provides a stable point to attach orthodontic appliances, such as wires, springs, or elastic bands.
  3. Tooth Movement: The TAD helps direct precise forces to move specific teeth or groups of teeth, allowing for complex movements like closing gaps, correcting bite issues, or aligning teeth more effectively.
  4. Removal: After the desired tooth movement is achieved, the TAD is easily removed, and the area heals quickly.

Why Are TADs Used?

TADs are used to address a variety of orthodontic challenges that might be difficult or impossible to achieve with braces or aligners alone. Some common uses include:

  • Closing Large Gaps: TADs can help close spaces between teeth, such as those caused by missing teeth.
  • Correcting Severe Misalignments: They assist in moving teeth that are significantly out of position.
  • Improving Bite Issues: TADs are often used to correct overbites, underbites, or open bites.
  • Reducing the Need for Extractions: By providing precise control, TADs can sometimes eliminate the need to remove teeth to create space.
  • Supporting Complex Cases: They’re especially helpful in adult orthodontics or cases where traditional methods are less effective.

Benefits of TADs

TADs offer several advantages that make them a valuable tool in orthodontics:

  • Precision: They allow for highly controlled tooth movements, leading to more predictable results.
  • Efficiency: TADs can reduce treatment time by enabling faster and more direct tooth movements.
  • Minimally Invasive: Placement and removal are quick and typically involve minimal discomfort.
  • Versatility: They can be used in a wide range of cases, from minor adjustments to complex treatments.
  • Reduced Reliance on Patient Compliance: Unlike headgear or elastics, which depend on consistent wear, TADs work without requiring extra effort from the patient.

What to Expect During TAD Placement

If your orthodontist recommends TADs, here’s what you can expect:

  • The Procedure: The area where the TAD will be placed is numbed with local anesthesia, so you’ll feel little to no pain. Some patients report mild pressure during insertion. The process is quick, often taking less than 10 minutes.
  • After Placement: You may experience mild soreness or sensitivity for a day or two, similar to what you feel after getting braces adjusted. Over-the-counter pain relievers, like ibuprofen, can help if needed.
  • Care Instructions: Keeping the area clean is important to prevent irritation or infection. Your orthodontist will provide guidance on brushing gently around the TAD and avoiding hard or sticky foods that could dislodge it.
  • Duration: TADs are typically left in place for a few months to a year, depending on your treatment plan. Once their job is done, they’re removed in a simple procedure, and the gum tissue heals quickly.


Are There Any Risks?

TADs are generally safe, but like any dental procedure, there are minor risks, including:

  • Mild Discomfort: Some soreness or irritation around the TAD site is normal but usually temporary.
  • Loosening: In rare cases, a TAD may become loose and need repositioning or replacement.
  • Infection: Proper oral hygiene minimizes this risk, but it’s important to follow your orthodontist’s care instructions.

Your orthodontist will discuss these risks and ensure TADs are a good fit for your treatment.

Frequently Asked Questions

Will TADs hurt?
The placement is done under local anesthesia, so you won’t feel pain during the procedure. Any post-placement discomfort is typically mild and short-lived.

Will TADs change my appearance?
TADs are small and placed in discreet areas of the mouth, so they’re usually not noticeable when you smile or talk.

How do I care for TADs?
Brush gently around the TAD to keep the area clean, and avoid chewing hard or sticky foods near the device. Your orthodontist will provide specific instructions.

Can anyone get TADs?
TADs are suitable for many patients, but your orthodontist will evaluate factors like bone density and oral health to determine if they’re right for you.



Conclusion

Temporary Anchorage Devices are a game-changer in orthodontics, offering a precise, efficient, and minimally invasive way to achieve a beautiful, healthy smile. If your orthodontist suggests TADs, rest assured they’re a safe and effective tool to enhance your treatment. Feel free to ask your orthodontist any questions to better understand how TADs will work in your unique case. With TADs, you’re one step closer to the smile you’ve always wanted!

Which anti tubercular drug crosses the blood brain barrier (BBB)?

 # Which anti tubercular drug crosses the blood brain barrier (BBB)?
A. INH
B. Rifampicin
C. Ethambutol
D. Streptomycin


The correct answer is A. INH (Isoniazid).

Explanation:
INH (Isoniazid): 
- Crosses the blood-brain barrier (BBB) effectively, even in the absence of inflammation. 
- First-line drug for tuberculous meningitis due to excellent CSF penetration.
- Critical for treating CNS tuberculosis.

Other Options:
B. Rifampicin: Penetrates the BBB only when meninges are inflamed (e.g., in active meningitis). Not as reliable as INH under normal conditions.
C. Ethambutol and D. Streptomycin: Poor BBB penetration, making them unsuitable for CNS tuberculosis.
Key Takeaway:
INH is the most reliable anti-tubercular drug for crossing the BBB, especially in latent or early CNS infections. Rifampicin’s efficacy depends on meningeal inflammation.

Which of the following can be diagnosed using dark field microscopy?

 # Which of the following can be diagnosed using dark field microscopy?
A. Spirochaetes
B. Streptococci
C. Corynebacteria
D. Mycobacteria


The correct answer is A. Spirochaetes.

Explanation:

Dark field microscopy is particularly useful for observing organisms that are difficult to stain, such as Spirochaetes (e.g., Treponema pallidum, the causative agent of syphilis). These bacteria are thin and motile, making dark field microscopy ideal for visualizing their morphology and movement.

Streptococci (B), Corynebacteria (C), and Mycobacteria (D) are typically diagnosed using Gram staining, Albert staining, or acid-fast staining, respectively. They do not require dark field microscopy.

Lateral Cephalogram Samples for Analysis

 These are some samples of Lateral cephalograms you can use to study and do analyses.